Dioxin-induced immortalization of normal human keratinocytes and silencing of p53 and p16INK4a.

Cytochrome P450 expression in human keratinocytes: an aryl hydrocarbon receptor perspective.

The p53 inhibitor pifithrin-alpha is a potent agonist of the aryl hydrocarbon receptor.

Role of arginine residues 14 and 15 in dictating DNA binding stability and transactivation of the aryl hydrocarbon receptor/aryl hydrocarbon receptor nuclear translocator heterodimer.

Role of arginine residues 14 and 15 in dictating DNA binding stability and transactivation of the aryl hydrocarbon receptor/aryl hydrocarbon receptor nuclear translocator heterodimer.

[Anonymous].  2005.  Role of arginine residues 14 and 15 in dictating DNA binding stability and transactivation of the aryl hydrocarbon receptor/aryl hydrocarbon receptor nuclear translocator heterodimer.. Gene expression. 12(4-6):231-43.

Identification of kaempferol as an inhibitor of cigarette smoke-induced activation of the aryl hydrocarbon receptor and cell transformation.

Identification of kaempferol as an inhibitor of cigarette smoke-induced activation of the aryl hydrocarbon receptor and cell transformation.

[Anonymous].  2007.  Identification of kaempferol as an inhibitor of cigarette smoke-induced activation of the aryl hydrocarbon receptor and cell transformation.. Carcinogenesis. 28(3):639-47.

Lack of the aryl hydrocarbon receptor leads to impaired activation of AKT/protein kinase B and enhanced sensitivity to apoptosis induced via the intrinsic pathway.

Lack of the aryl hydrocarbon receptor leads to impaired activation of AKT/protein kinase B and enhanced sensitivity to apoptosis induced via the intrinsic pathway.

[Anonymous].  2007.  Lack of the aryl hydrocarbon receptor leads to impaired activation of AKT/protein kinase B and enhanced sensitivity to apoptosis induced via the intrinsic pathway.. The Journal of pharmacology and experimental therapeutics. 320(1):448-57.

Cigarette smoke condensate and dioxin suppress culture shock induced senescence in normal human oral keratinocytes.

Development of an aryl hydrocarbon receptor antagonist using the proteolysis-targeting chimeric molecules approach: a potential tool for chemoprevention.

Development of an aryl hydrocarbon receptor antagonist using the proteolysis-targeting chimeric molecules approach: a potential tool for chemoprevention.

[Anonymous].  2008.  Development of an aryl hydrocarbon receptor antagonist using the proteolysis-targeting chimeric molecules approach: a potential tool for chemoprevention.. Molecular pharmacology. 73(4):1064-71.

Activation of the aryl hydrocarbon receptor by TCDD inhibits senescence: a tumor promoting event?

Targeting drug-metabolizing enzymes for effective chemoprevention and chemotherapy.