The mitochondrial uncoupling agent 2,4-dinitrophenol improves mitochondrial function, attenuates oxidative damage, and increases white matter sparing in the contused spinal cord.

The mitochondrial uncoupling agent 2,4-dinitrophenol improves mitochondrial function, attenuates oxidative damage, and increases white matter sparing in the contused spinal cord.

[Anonymous].  2004.  The mitochondrial uncoupling agent 2,4-dinitrophenol improves mitochondrial function, attenuates oxidative damage, and increases white matter sparing in the contused spinal cord.. Journal of neurotrauma. 21(10):1396-404.

Overexpression of XIAP inhibits apoptotic cell death in an oligodendroglial cell line.

Neuroprotection and acute spinal cord injury: a reappraisal.

A mapping study of caspase-3 activation following acute spinal cord contusion in rats.

Apoptotic cell death following traumatic injury to the central nervous system.

Quantification of locomotor recovery following spinal cord contusion in adult rats.

Pretreatment with the cyclosporin derivative, NIM811, improves the function of synaptic mitochondria following spinal cord contusion in rats.

Pretreatment with the cyclosporin derivative, NIM811, improves the function of synaptic mitochondria following spinal cord contusion in rats.

[Anonymous].  2007.  Pretreatment with the cyclosporin derivative, NIM811, improves the function of synaptic mitochondria following spinal cord contusion in rats.. Journal of neurotrauma. 24(4):613-24.

Post-treatment with the cyclosporin derivative, NIM811, reduced indices of cell death and increased the volume of spared tissue in the acute period following spinal cord contusion.

Post-treatment with the cyclosporin derivative, NIM811, reduced indices of cell death and increased the volume of spared tissue in the acute period following spinal cord contusion.

[Anonymous].  2007.  Post-treatment with the cyclosporin derivative, NIM811, reduced indices of cell death and increased the volume of spared tissue in the acute period following spinal cord contusion.. Journal of neurotrauma. 24(10):1618-30.

Attenuation of acute mitochondrial dysfunction after traumatic brain injury in mice by NIM811, a non-immunosuppressive cyclosporin A analog.

Attenuation of acute mitochondrial dysfunction after traumatic brain injury in mice by NIM811, a non-immunosuppressive cyclosporin A analog.

[Anonymous].  2008.  Attenuation of acute mitochondrial dysfunction after traumatic brain injury in mice by NIM811, a non-immunosuppressive cyclosporin A analog.. Experimental neurology. 209(1):243-53.

Focal cerebral ischemia in the TNFalpha-transgenic rat.